Heart disease in an otherwise healthy young athlete who is abusing anabolic steroids likely results from increased levels of low-density lipoprotein (LDL) and decreased levels of high-density lipoprotein (HDL).
Several studies suggest that anabolic steroid abuse in athletes increases LDL levels by > 20% and decreases HDL levels by 20% to 70%. These lipoprotein abnormalities have been shown to increase the risk for coronary artery disease three- to six-fold. Anabolic steroid abuse may cause cardiac ischemia by exaggerating oxygen demand at peak exercise, potentially precipitated by accelerated atherosclerosis from lipoprotein abnormalities over years of abuse.
Silver reviewed the adverse effects of anabolic steroids in a JAAOS article and reported that anabolic steroid use can lead to hypertension, changes in lipid profile, elevated liver enzymes, increased risk of tendon and muscle injury, testicular or uterine atrophy, depression, psychosis, and immunosuppression.
Achar et al. reviewed a total of 49 studies describing 1,467 athletes to investigate the cardiovascular effects of the anabolic steroid abuse. They found that anabolic steroid abuse was associated with elevated levels of LDL, low levels of HDL, elevated systolic and diastolic blood pressure, and left ventricular hypertrophy. They also noted that there were some small case studies describing fatal ventricular arrhythmias secondary to anabolic steroid abuse.
Answer 1: High density lipoprotein levels are decreased, not increased, in those abusing anabolic steroids. This decrease in HDL levels contributea to the development of heart disease in these otherwise healthy individuals.
Answer 2: Systolic and diastolic blood pressure are elevated, not decreased, after prolonged anabolic steroid use. This does not lead to heart disease in those who abuse anabolic steroids.
Answer 4: Alteration of myocardial conduction patterns (., ventricular arrhythmia) is not a cause of heart disease in anabolic steroid abusers. It is, however, a cause of sudden death in these individuals.
Answer 5: Anabolic steroids do not cause 'direct' endothelial damage to the coronary arteries.
It is very common for bodybuilders to use veterinary steroids for a precontest cycle. Since they are typically assimilated quickly, they do the best work in the shortest amount of time, and are generally out of the system relatively fast in comparison to other ‘roids. And, believe it or not, usually people see fewer side effects when using vet products than when using human ones. Why take anything else? Maybe not ‘why take anything else’, but why not include veterinary steroids, in one form or another, in every cycle? In my mind, veterinary drugs should really be everyone’s choice for extreme condition and definition. They combine well with androgens and other anabolics as well as any drug in the human realm of anabolic steroids. The only problem, these days anyway, is availability. You can find them in Mexico, but you risk fakes, counterfeits, lower quality, or lower dose per ml. You also, of course, face the possibility that you’ll be stopped.
The most commonly used AAS in medicine are testosterone and its various esters (but most commonly testosterone undecanoate , testosterone enanthate , testosterone cypionate , and testosterone propionate ),  nandrolone esters (most commonly nandrolone decanoate and nandrolone phenylpropionate ), stanozolol , and metandienone (methandrostenolone).  Others also available and used commonly but to a lesser extent include methyltestosterone , oxandrolone , mesterolone , and oxymetholone , as well as drostanolone propionate , metenolone (methylandrostenolone), and fluoxymesterone .  Dihydrotestosterone (DHT; androstanolone, stanolone) and its esters are also notable, although they are not widely used in medicine.  Boldenone undecylenate and trenbolone acetate are used in veterinary medicine .